Occurrence of mitochondria-targeted Late Embryogenesis Abundant (LEA) gene in animals increases organelle resistance to water stress.
Document Type
Article
Publication Date
1-1-2009
Department
Biology
Abstract
Anhydrobiotic animals survive virtually complete loss of cellular water. The mechanisms that explain this phenomenon are not fully understood but often include the accumulation of low molecular weight solutes such as trehalose and macromolecules like Late Embryogenesis Abundant (LEA) proteins. Here we report for the first time the occurrence of a mitochondria-targeted LEA gene (Afrlea3m) product in an animal species.
ThinkIR Citation
Menze, Michael; Boswell, Leaf; Toner, Mehmet; and Hand, Steven, "Occurrence of mitochondria-targeted Late Embryogenesis Abundant (LEA) gene in animals increases organelle resistance to water stress." (2009). Faculty Scholarship. 104.
https://ir.library.louisville.edu/faculty/104
Comments
This research was originally published in the Journal of Biological Chemistry. Menze, M. A., Boswell, L., Toner, M., and Hand, S. C. Occurrence of mitochondrial-targeted late embryogenesis abundant (LEA) gene in animals increases organelle resistance to water stress. Journal of Biological Chemistry. 2009. 284, 16:10714-10719. © the American Society for Biochemistry and Molecular Biology